Medicament for treating tinea cruris and preparation method

Abstract

The invention discloses a medicament for treating tinea cruris and a preparation method. The medicament is characterized by being prepared by the following raw materials by weight ratio: 10-12 parts of sophora flavescens, 20-60 parts of fructus kochiae, 30-50 parts of impatiens balsamina herb, 30-50 parts of shrubalthea bark, 10-50 parts of rhizoma atractylodis, 10-50 parts of phellodendron, 10-50 parts of wormseed, 10-40 parts of sweet wormwood, 10-60 parts of alum, 10-60 parts of borax, 10-50 parts of common cnidium fruit, 10-50 parts of dictamnus dasycarpus turcz, 10-40 parts of pericarpium zanthoxyli, 60-80 parts of radix aristolochiae, 20-50 parts of stemona, 10-20 parts of tarragon, 5-10 parts of pericarpium zanthoxyli, 20-40 parts of coptis, 40-80 parts of dried alum, 12-14 parts of borneol , and 10-60 parts of vinegar.

Description

Technical field: [0001] The invention relates to a medicine for treating tinea corporis and a preparation method thereof. Background technique: [0002] Tinea corporis is a general term for tinea corporis and jock itch. Tinea corporis is a dermatophyte infection of the skin of the trunk and extremities, excluding the hair, palms and soles, nails, and groin. It is endemic worldwide, most commonly in tropical regions. Can be transmitted from person to person, animal to person, and soil to person. Tinea corporis is the most common cause of domestic animals, and other predisposing factors include personal presence of tinea capitis or tinea pedis, close contact with an ill person, exposure to contaminated clothing or furniture, immunosuppression, occupational or recreational exposure, etc. Usually occurs in exposed areas, but can spread to any part of the body surface. Jock itch is a dermatophyte infection of the groin, especially the inner thigh and creases, often involving ...

AI Technical Summary

Problems solved by technology

However, long-term use of antibiotics can cause the imbalance of flora in the body. When the skin of the human body is damaged and the resistance decreases, the pathogenic molds will multiply and invade the skin and subcutaneous tissue to cause ringworm.